anticol3a1 primary antibodies Search Results


col3a1 protein 1:1000 antibody  (GeneTex)
90
GeneTex col3a1 protein 1:1000 antibody
Differential gene expressions between healthy tenocyte and tendinopathy tenocyte. Relative expressions by qRT-PCR of 8 genes were analyzed using cultured tenocytes. Results show that levels of <t>COL3A1</t> , MMP1 , COX2 , SCX , ACTA2 , TAC1 ( SP ), and NK-1R were significantly increased in the cells cultured from tendinopathy tenocyte. However, COL1A1 was decreased in the cells cultured from tendinopathy tenocyte without statistical significance. The result was expressed as the relative expression level. Data were normalized on GAPDH mRNA levels. Boxplot showing the median along with the first and third quartiles. Error bar means data range from minimum to maximum. ** p < 0.01. SP: substance P; qRT-PCR: quantitative real time-polymerase chain reaction results.
Col3a1 Protein 1:1000 Antibody, supplied by GeneTex, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/anticol3a1+primary+antibodies/pmc05486064-145-6-9?v=GeneTex
Average 90 stars, based on 1 article reviews
col3a1 protein 1:1000 antibody - by Bioz Stars, 2026-08
90/100 stars
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anti col3a1  (Santa Cruz Biotechnology)
94
Santa Cruz Biotechnology anti col3a1
Differential gene expressions between healthy tenocyte and tendinopathy tenocyte. Relative expressions by qRT-PCR of 8 genes were analyzed using cultured tenocytes. Results show that levels of <t>COL3A1</t> , MMP1 , COX2 , SCX , ACTA2 , TAC1 ( SP ), and NK-1R were significantly increased in the cells cultured from tendinopathy tenocyte. However, COL1A1 was decreased in the cells cultured from tendinopathy tenocyte without statistical significance. The result was expressed as the relative expression level. Data were normalized on GAPDH mRNA levels. Boxplot showing the median along with the first and third quartiles. Error bar means data range from minimum to maximum. ** p < 0.01. SP: substance P; qRT-PCR: quantitative real time-polymerase chain reaction results.
Anti Col3a1, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/anticol3a1+primary+antibodies/pmc04196650-62-27-34?v=Santa+Cruz+Biotechnology
Average 94 stars, based on 1 article reviews
anti col3a1 - by Bioz Stars, 2026-08
94/100 stars
  Buy from Supplier
anticol3a1 primary antibodies  (Proteintech)
96
Proteintech anticol3a1 primary antibodies
Differential gene expressions between healthy tenocyte and tendinopathy tenocyte. Relative expressions by qRT-PCR of 8 genes were analyzed using cultured tenocytes. Results show that levels of <t>COL3A1</t> , MMP1 , COX2 , SCX , ACTA2 , TAC1 ( SP ), and NK-1R were significantly increased in the cells cultured from tendinopathy tenocyte. However, COL1A1 was decreased in the cells cultured from tendinopathy tenocyte without statistical significance. The result was expressed as the relative expression level. Data were normalized on GAPDH mRNA levels. Boxplot showing the median along with the first and third quartiles. Error bar means data range from minimum to maximum. ** p < 0.01. SP: substance P; qRT-PCR: quantitative real time-polymerase chain reaction results.
Anticol3a1 Primary Antibodies, supplied by Proteintech, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/anticol3a1+primary+antibodies/pm39127300-114-34-38?v=Proteintech
Average 96 stars, based on 1 article reviews
anticol3a1 primary antibodies - by Bioz Stars, 2026-08
96/100 stars
  Buy from Supplier

Image Search Results


Differential gene expressions between healthy tenocyte and tendinopathy tenocyte. Relative expressions by qRT-PCR of 8 genes were analyzed using cultured tenocytes. Results show that levels of COL3A1 , MMP1 , COX2 , SCX , ACTA2 , TAC1 ( SP ), and NK-1R were significantly increased in the cells cultured from tendinopathy tenocyte. However, COL1A1 was decreased in the cells cultured from tendinopathy tenocyte without statistical significance. The result was expressed as the relative expression level. Data were normalized on GAPDH mRNA levels. Boxplot showing the median along with the first and third quartiles. Error bar means data range from minimum to maximum. ** p < 0.01. SP: substance P; qRT-PCR: quantitative real time-polymerase chain reaction results.

Journal: International Journal of Molecular Sciences

Article Title: The Implication of Substance P in the Development of Tendinopathy: A Case Control Study

doi: 10.3390/ijms18061241

Figure Lengend Snippet: Differential gene expressions between healthy tenocyte and tendinopathy tenocyte. Relative expressions by qRT-PCR of 8 genes were analyzed using cultured tenocytes. Results show that levels of COL3A1 , MMP1 , COX2 , SCX , ACTA2 , TAC1 ( SP ), and NK-1R were significantly increased in the cells cultured from tendinopathy tenocyte. However, COL1A1 was decreased in the cells cultured from tendinopathy tenocyte without statistical significance. The result was expressed as the relative expression level. Data were normalized on GAPDH mRNA levels. Boxplot showing the median along with the first and third quartiles. Error bar means data range from minimum to maximum. ** p < 0.01. SP: substance P; qRT-PCR: quantitative real time-polymerase chain reaction results.

Article Snippet: We used the following primary antibodies: COL3A1 protein 1:1000 (Gene Tex, Irvine, CA, USA), COX2 protein 1:1000 (Gene Tex), and MMP1 protein 1:1000 (Gene Tex).

Techniques: Quantitative RT-PCR, Cell Culture, Expressing, Real-time Polymerase Chain Reaction

SP upregulation of genes related to tendinopathy. To determine the effects of SP on the development of tendinopathy, cultured healthy tenocytes were pretreated with or without SP 10 −7 M for 24 h. Results show that healthy tenocytes developed similar gene expression trends as that of the tendinopathy tenocyte. Levels of COL1A1 decreased but COL3A1 , COX2 , SCX , MMP1 , and ACTA2 were significantly increased as a result of treatment with SP. The result was expressed as the relative expression level after normalizing on GAPDH mRNA levels. Boxplot showing the median along with the first and third quartiles. Error bar means data range from minimum to maximum. * p < 0.05; ** p < 0.01; SP: substance P.

Journal: International Journal of Molecular Sciences

Article Title: The Implication of Substance P in the Development of Tendinopathy: A Case Control Study

doi: 10.3390/ijms18061241

Figure Lengend Snippet: SP upregulation of genes related to tendinopathy. To determine the effects of SP on the development of tendinopathy, cultured healthy tenocytes were pretreated with or without SP 10 −7 M for 24 h. Results show that healthy tenocytes developed similar gene expression trends as that of the tendinopathy tenocyte. Levels of COL1A1 decreased but COL3A1 , COX2 , SCX , MMP1 , and ACTA2 were significantly increased as a result of treatment with SP. The result was expressed as the relative expression level after normalizing on GAPDH mRNA levels. Boxplot showing the median along with the first and third quartiles. Error bar means data range from minimum to maximum. * p < 0.05; ** p < 0.01; SP: substance P.

Article Snippet: We used the following primary antibodies: COL3A1 protein 1:1000 (Gene Tex, Irvine, CA, USA), COX2 protein 1:1000 (Gene Tex), and MMP1 protein 1:1000 (Gene Tex).

Techniques: Cell Culture, Gene Expression, Expressing

SP induced the development of tendinopathy at protein level. Western blot for the cultured healthy tenocyte with or without SP was performed with antibodies against COL3A1 , COX2 , and MMP1 protein. Loading control was performed with an anti-β-actin antibody. Twenty-four hours of exposure to SP resulted in strong band densities for every protein. The results were consistent with the analysis of qRT-PCR ( A ); Densitometric analysis, as expected, revealed that COL3A1 , COX2 , and MMP1 protein band intensities normalized to β-actin were increased by 203.8%, 129.3%, and 129.3% respectively in SP-treated group compared with PBS-control. Boxplot showing the median along with the first and third quartiles ( B ). Error bar means data range from minimum to maximum. * p < 0.05; SP: substance P.

Journal: International Journal of Molecular Sciences

Article Title: The Implication of Substance P in the Development of Tendinopathy: A Case Control Study

doi: 10.3390/ijms18061241

Figure Lengend Snippet: SP induced the development of tendinopathy at protein level. Western blot for the cultured healthy tenocyte with or without SP was performed with antibodies against COL3A1 , COX2 , and MMP1 protein. Loading control was performed with an anti-β-actin antibody. Twenty-four hours of exposure to SP resulted in strong band densities for every protein. The results were consistent with the analysis of qRT-PCR ( A ); Densitometric analysis, as expected, revealed that COL3A1 , COX2 , and MMP1 protein band intensities normalized to β-actin were increased by 203.8%, 129.3%, and 129.3% respectively in SP-treated group compared with PBS-control. Boxplot showing the median along with the first and third quartiles ( B ). Error bar means data range from minimum to maximum. * p < 0.05; SP: substance P.

Article Snippet: We used the following primary antibodies: COL3A1 protein 1:1000 (Gene Tex, Irvine, CA, USA), COX2 protein 1:1000 (Gene Tex), and MMP1 protein 1:1000 (Gene Tex).

Techniques: Western Blot, Cell Culture, Control, Quantitative RT-PCR